Paper Details 
Original Abstract of the Article :
The gold compounds, auranofin, sodium aurothiomalate, and triethyl gold phosphine have been demonstrated to inhibit various effector functions associated with monocyte-macrophage populations. Incubation of human peripheral blood monocytes and murine peritoneal macrophages with auranofin or triethyl ...See full text at original site
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ラクダ博士は、Health Journal が論文の内容を分かりやすく解説するために作成した架空のキャラクターです。
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* ラクダ博士は架空のキャラクターであり、実際の医学研究者や医療従事者とは一切関係がありません。
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引用元:
https://doi.org/10.1007/BF01967297

データ提供:米国国立医学図書館(NLM)

Gold Compounds: A Potential Oasis in the Desert of Inflammation

Inflammation is a complex and often debilitating process, like a relentless sandstorm that can erode the body's health. This study focuses on gold compounds, a group of substances known for their potential anti-inflammatory properties. The researchers aimed to understand how these compounds affect the production of TNF, a powerful inflammatory molecule that can contribute to a range of diseases.

TNF Production: A Key Player in Inflammation

TNF is like a desert wind that whips up a storm of inflammation. The study found that auranofin and other gold compounds effectively inhibited TNF production in both laboratory models and live animals. This suggests that these compounds might act as a calming force in the desert of inflammation, preventing the storm from escalating.

New Directions for Treating Chronic Inflammation

The study's findings provide a glimmer of hope for those struggling with chronic inflammatory diseases. Gold compounds, like a refreshing spring in the desert, could offer a new approach to managing these conditions. However, as with any new treatment, further research is needed to determine the safety and efficacy of these compounds in the long-term.

Dr. Camel's Conclusion

This study explores the potential of gold compounds as anti-inflammatory agents, offering a potential oasis in the desert of chronic inflammation. Their ability to inhibit TNF production suggests a promising avenue for future therapeutic development.

Date :
  1. Date Completed 1989-07-18
  2. Date Revised 2019-08-24
Further Info :

Pubmed ID

2500010

DOI: Digital Object Identifier

10.1007/BF01967297

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Languages

English

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